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With the overload supply of metabolic substrates, excessive fat accumulates in the hepatocytes, accompanied by gradual generation of potential toxic lipid species and the progressive increase in de novo lipogenesis (DNL) in NAFLD (117)

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Instead, their decreased food purchases are disproportionately concentrated in processed foods and sugar-sweetened beverages, further underscoring the shift in food preference induced by GLP-1R-based treatments (Dilley et al., 2025)