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High-quality randomized controlled trials reported in both Chinese and English were included in this analysis

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Conclusion In conclusion, our findings demonstrate that ROS regulate GSK3 dephosphorylation via the PI3K/Akt pathway, which in turn activates IP3R on the ER membrane to induce the release Ca 2+ into the cytoplasm

The mechanistic benefits include increased fibroblast activity, improved skin elasticity, enhanced wound healing capacity, and better skin barrier function through ceramide production