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39 Additionally, there is established evidence of alterations in glutamate circuits and GABAergic circuits in ASD patients, as manifested by increased numbers of excitatory synapses and spine densities, significantly reduced levels of glutamic acid decarboxylase, and GABAA and GABAB receptor alterations in the postmortem brains of patients with autism
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Glucagon receptor activation is examined for its relationship with hepatic glucose output, lipolysis, and energy-expenditure pathways central to weight loss research
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