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In line with the concept that T cell activation and differentiation require metabolic adaptation, SLC1A5 couples TCR and CD28 signals to ultimately activate the mTORC1 pathway and enable the cell to undergo clonal expansion (105)

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Additionally, GSTO1-1 has been shown to modulate Akt and MEK1/2 kinase pathways in human neuroblastoma SH-SY5Y cells, where the catalytic activities of the isoenzyme was shown to suppress the activation of these two kinases (either directly or in a complex) to maintain basal levels [69]

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