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An excessive buildup of ROS drives oxidative cell death

Optimization of the hemolysis assay for the assessment of cytotoxicity

Like Th1 cells, also Th17 cells promote inflammation in MS, transmigrating across Blood-Brain Barrier (BBB) and may induce demyelination and axonal loss (Moser et al., 2020)

Several pieces of scientific evidence support this

Its a highly effective scavenger of free radicals and other reactive oxygen species (ROS) and reactive nitrogen species (RNS) such as the hydroxyl radical, lipid peroxyl radical, superoxide anion, and hydrogen peroxide, both directly and indirectly through enzymatic reactions 5

The crosstalk between distinct RCDs is well documented and can be interpreted through the lens of the cell death-governing intracellular system with multiple redundant backup pathways [20, 142]