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GPx6, the last among the seleno-GPx family members, is expressed exclusively in the adult olfactory epithelium [1, 8]

Key molecular mechanisms encompass oxidative modification of critical proteins leading to dysfunction [361], membrane lipid peroxidation disrupting organelle integrity [362], mtDNA damage causing mitochondrial dysfunction [363], and depletion of key antioxidant molecules [364]

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Interestingly, the elevated CHAC1 levels in TP53-mutant cells might represent an adaptive response, potentially compensating for defective ferroptosis pathways or preventing harmful GSH accumulation

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