Environmental toxins, chronic stress, poor sleep quality, excessive alcohol consumption, and certain medications all play a role
Jimenez Trinidad FR, Arrieta Ruiz M, Solanes Batllo N, Vea Badenes A, Bobi Gibert J, Valera Canellas A, Roque Moreno M, Freixa Rofastes X, Sabate Tenas M, Dantas AP, Tura-Ceide O, Rigol Muxart M (2021) Linking in vitro models of endothelial dysfunction with cell senescence
NOT for clinical, diagnostic, or therapeutic purposes
Normally, our bodies should make enough glutathione to protect us
The oxidative damage mediated by ROS resulting in the defect in control of apoptosis or programmed cell death and delayed clearance of apoptotic cells may prolong interaction between ROS and apoptotic cell macromolecules generating neoepitopes that subsequently broad spectrum of autoantibody formation leading to the tissue damage in SLE [6,7]
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