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9,10 Mitochondria, which are critical for cellular respiration and metabolism, suffer damage to their own mitochondrial DNA by the actions of reactive oxygen species and peroxynitrite compounds, and they have been directly implicated in the process leading to cessation of ATP synthesis

3a) and therefore the N-terminal dipeptide is removed by DPP-4

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CaMK4-dependent activation of AKT/mTOR and CREM- underlies autoimmunity-associated Th17 imbalance

ME wrote the article, XG wrote and critically reviewed the article and EI wrote and critically reviewed the article

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