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227,228 Immunomodulation HGF-overexpressing MSCs promote the differentiation of mature DCs (mDCs) into tolerogenic dendritic cells (DCregs) via direct cellcell contact, thus reducing ALI through the HGF/Akt pathway and the Notch pathway

Postprandial production of GLP-1 increases insulin secretion from pancreatic beta cells and suppresses glucagon secretion from pancreatic alpha cells

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Mitochondria related cell death modalities and disease

Keywords: Diabetes mellitus, Diabetes complications, Glucagon-like peptide 1 receptor agonists, Sodium-glucose cotransporter 2 inhibitors Key Summary Points Introduction Individuals with type 2 diabetes (T2D) face a heightened risk of cardiovascular and cerebrovascular (CV) events, alongside increased mortality

We considered a subject to have a positive response to GLP-1 M if the median HbA1C within 12 months after the treatment decreased by more than 0.5% from the median HbA1C within 12 months before the treatment [13, 14]