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Endoplasmic reticulum stress and oxidative stress The accumulation of lipotoxic speciesincluding free fatty acids (FFAs), diacylglycerols, phospholipids, and free cholesteroltriggers endoplasmic reticulum (ER) stress, leading to activation of multiple cellular stress pathways

This time, selenocysteine also becomes a key factor along with seleno-homocysteine

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[DOI] [PMC free article] [PubMed] [Google Scholar] 99.Fras JP, Davies MJ, Rosenstock J, et al

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