Jansson et al., 2022), reducing resting and ambulatory blood pressure in individuals with hypertension (Correia et al., 2023), enhancing muscular strength and function in sarcopenia (Zhao et al., 2022) and osteoarthritis (Lim et al., 2024), and positively influencing vascular health in those with cardiovascular disease (Paluch et al., 2024
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However, under defined pathophysiological stresses, including inflammatory cues, metal overload, cysteine deprivation, or drug exposure, GSH oxidation and thiol chemistry may become coupled to metal redox cycling and mitochondrial damage amplification, thereby increasing susceptibility to forms of regulated cell death such as ferroptosis and cuproptosis
doi:10.1503/cmaj.181577
Activated microglia migrate towards senile plaques, engulf A, and release enzymes to break down A
GSH as intracellular copper chelator The most fundamental protective mechanism of GSH against cuproptosis is its function as an intracellular copper chelator
