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For people with MTHFR mutations, synthetic folic acid can actually block folate receptors and worsen symptoms

The same logic that makes MOTS-C work with Retatrutide applies to semaglutide and tirzepatide, just to a lesser degree

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Compared to healthy controls, patients with T2D have lower levels of red blood cell GSH levels as well as lower GSH synthesis rates, particularly in T2D patients with microvascular complications [8]

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