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It is worth noting that we used TAT peptide as the control peptide in this study rather than the second N-terminus fragment of DAT (A 16 P 26 ) from our previous study [23]

The overexpression of circ_0000250 in ADSC-EVs results in enhanced healing in diabetic mice through the induction of autophagy mediated by the miR-128-3p/SIRT1 axis since epidermal SIRT1 is responsible for the modulation of inflammation, wound healing and cell migration [120]