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Further mechanistic studies revealed that LPS downregulates UFL1 expression, activates p53/SLC7A11 system, and modulates autophagy, thereby inducing ferroptosis (60)

Matrix metalloproteinases (MMPs), particularly MMP-1 and MMP-2, actively break down existing collagen

Completed human trials establishing safety and efficacy would enable consideration for approval pathways

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The peptide's effects were mediated through the downregulation of miR-339-5p expression, which was found to be part of the p38 MAPK pathway

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