C-terminal amidation, as seen in Nle-Tyr-Ile-His-NH, also provided a more modest increase in stability
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As erythrocytes from patients with inherited enzyme deficiencies (e.g., glucose-6-phosphate dehydrogenase-deficient cells) are more susceptible to different inducers of eryptosis, it seems reasonable to mention RBC enzyme deficiency-dependent erythrocyte death [80]
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Eryptosis and occupational exposure to lead An epidemiological study of male workers exposed to lead (64.8 g lead/dL blood) and workers not exposed to lead (4.2 g lead/dL blood) revealed a poisoning, characterized by 88.3% lower -aminolevulinic acid dehydratase (ALAD) activity