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FOXO4-DRI enters senescent cells and competitively binds to p53 through its modified peptide sequence, displacing endogenous FOXO4 and freeing p53 to activate cell death pathways including BAX and PUMA

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This prevents bacterial growth, allowing the solution to remain sterile for up to 28 days after opening

Oxidative Stress Response GHK-Cu demonstrates potent ROS reduction in cell cultures[21]

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