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The primary cause of glucotoxicity is insulin resistance, which arises from defective islet cell function and damage caused by reduced insulin mRNA expression, associated with diminished transcription or activity of transcription factors involved in insulin production (Danil and Michala, 2011)

The trial, which enrolled 416 participants with early Alzheimer's disease, did not meet its primary endpoint assessing dose response on the Clinical Dementia RatingSum of Boxes at week 76

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Extended bioavailability compared to native IGF-1 due to reduced binding protein interaction [2]